Publications

Journal Articles

  1. & Massive Accumulation of Myofibroblasts in the Critical Isthmus Is Associated With Ventricular Tachycardia Inducibility in Post-Infarct Swine Heart. JACC: Clinical Electrophysiology 3(7), 703-714.
  2. & A Systemized Approach to Investigate Ca2+ Synchronization in Clusters of Human Induced Pluripotent Stem-Cell Derived Cardiomyocytes. Frontiers in Cell and Developmental Biology 3
  3. et. al. The vascular Ca2+-sensing receptor regulates blood vessel tone and blood pressure. American Journal of Physiology - Cell Physiology 310, ajpcell.00248.2015
  4. Local, integrated control of blood flow. Autonomic Neuroscience 178(1-2), 4-8.
  5. & The effect of inorganic arsenic on endothelium-dependent relaxation: Role of NADPH oxidase and hydrogen peroxide. Toxicology 306, 50-58.
  6. An investigation of the role of calcimimetics in a murine model of targeted deletion of the calcium-sensing receptor, CaSR, from vascular smooth muscle cells (SM22α x fl CaSR). Bone
  7. & Ascorbic acid and tetrahydrobiopterin potentiate the EDHF phenomenon by generating hydrogen peroxide. Cardiovascular Research 84(2), 218-226.
  8. & Attenuated store-operated Ca2+ entry underpins the dual inhibition of nitric oxide and EDHF-type relaxations by iodinated contrast media. Cardiovascular Research 84(3), 470-478.
  9. & Vasomotion: the case for chaos. Journal of Biorheology 23(1), 11-23.
  10. & Hydrogen Peroxide Potentiates the EDHF Phenomenon by Promoting Endothelial Ca2+ Mobilization. Arteriosclerosis, Thrombosis, and Vascular Biology 28(10), 1774-1781.
  11. & Modulation of Gap Junction-Dependent Arterial Relaxation by Ascorbic Acid. Journal of Vascular Research 44(5), 410-422.
  12. & What Do the Soldiers Say?. Armed Forces & Society 220(3), 367-388.
  13. & 5-Methyltetrahydrofolate and tetrahydrobiopterin can modulate electrotonically mediated endothelium-dependent vascular relaxation. Proceedings of the National Academy of Sciences 102(19), 7008-7013.
  14. & Connexin-mimetic peptides dissociate electrotonic EDHF-type signalling via myoendothelial and smooth muscle gap junctions in the rabbit iliac artery. British Journal of Pharmacology 144(1), 108-114.
  15. & Correction for Griffith et al., 5-Methyltetrahydrofolate and tetrahydrobiopterin can modulate electrotonically mediated endothelium-dependent vascular relaxation, PNAS 2005 102:7008-7013. Proceedings of the National Academy of Sciences 102(35), 12644-12644.
  16. & Enhanced inhibition of the EDHF phenomenon by a phenyl methoxyalaninyl phosphoramidate derivative of dideoxyadenosine. British Journal of Pharmacology 142(1), 27-30.
  17. & The obligatory link: role of gap junctional communication in endothelium-dependent smooth muscle hyperpolarization. Pharmacological Research 49(6), 551-564.
  18. & Shil'nikov homoclinic chaos is intimately related to type-III intermittency in isolated rabbit arteries: Role of nitric oxide. Physical Review E - Statistical, Nonlinear, and Soft Matter Physics 67
  19. & Distinct hyperpolarizing and relaxant roles for gap junctions and endothelium-derived H2O2 in NO-independent relaxations of rabbit arteries. Proceedings of the National Academy of Sciences 100(25), 15212-15217.
  20. & Endogenous Nitric Oxide Synthesis Differentially Modulates Pressure-Flow and Pressure-Conductance Relationships in the Internal and External Carotid Artery Circulations of the Rat. Neurologia medico-chirurgica 42(12), 527-535.
  21. & cAMP facilitates EDHF-type relaxations in conduit arteries by enhancing electrotonic conduction via gap junctions. Proceedings of the National Academy of Sciences 99(9), 6392-6397.
  22. & Universal scaling properties of type-I intermittent chaos in isolated resistance arteries are unaffected by endogenous nitric oxide synthesis. Physical Review E - Statistical, Nonlinear, and Soft Matter Physics 64
  23. & Gap junctional communication underpins EDHF-type relaxations evoked by ACh in the rat hepatic artery. American Journal of Physiology - Heart and Circulatory Physiology 280
  24. & Gap Junction-Dependent Increases in Smooth Muscle cAMP Underpin the EDHF Phenomenon in Rabbit Arteries. Biochemical and Biophysical Research Communications 283(3), 583-589.
  25. & Minimal model of arterial chaos generated by coupled intracellular and membrane Ca2+ oscillators. American Journal of Physiology - Heart and Circulatory Physiology 277
  26. & Integration of non-linear cellular mechanisms regulating microvascular perfusion. Proceedings of the Institution of Mechanical Engineers, Part H: Journal of Engineering in Medicine 213
  27. & Simplification of the quasiperiodic route to chaos in agonist-induced vasomotion by iterative circle maps. American Journal of Physiology - Heart and Circulatory Physiology 43
  28. & Simplification of the quasiperiodic route to chaos in agonist-induced vasomotion by iterative circle maps. American Journal of Physiology - Heart and Circulatory Physiology 274
  29. & Entrained ion transport systems generate the membrane component of chaotic agonist-induced vasomotion. American Journal of Physiology - Heart and Circulatory Physiology 273
  30. & Entrained ion transport systems generate the membrane component of chaotic agonist-induced vasomotion. American Journal of Physiology - Heart and Circulatory Physiology 42
  31. & Critical scaling and type-III intermittent chaos in isolated rabbit resistance arteries. Physical Review E - Statistical Physics, Plasmas, Fluids, and Related Interdisciplinary Topics 56
  32. & Ca2+ sequestration as a determinant of chaos and mixed-mode dynamics in agonist-induced vasomotion. American Journal of Physiology 272
  33. & Ca2+ sequestration as a determinant of chaos and mixed-mode dynamics in agonist-induced vasomotion. American Journal of Physiology - Heart and Circulatory Physiology 41
  34. & Ca2+ sequestration as a determinant of chaos and mixed-mode dynamics in agonist-induced vasomotion. American Journal of Physiology - Heart and Circulatory Physiology 272
  35. & Comparison of chaotic and sinusoidal vasomotion in the regulation of microvascular flow. Cardiovascular Research 31(3), 388-399.
  36. & Complexity of chaotic vasomotion is insensitive to flow and pressure but can be regulated by external control. American Journal of Physiology - Heart and Circulatory Physiology 269
  37. & Angiographic contrast media relax isolated rabbit aorta through an endothelium-independent mechanism that may not depend on the presence of the iodine atom. British Journal of Radiology 68
  38. & Fractal analysis of role of smooth muscle Ca2+ fluxes in genesis of chaotic arterial pressure oscillations. American Journal of Physiology - Heart and Circulatory Physiology 266
  39. & EDRF suppresses chaotic pressure oscillations in isolated resistance artery without influencing intrinsic complexity. American Journal of Physiology - Heart and Circulatory Physiology 266
  40. & Modulation of chaotic pressure oscillations in isolated resistance arteries by EDRF. European Heart Journal 14
  41. & Mechanisms underlying chaotic vasomotion in isolated resistance arteries: Roles of calcium and EDRF. Biorheology 30
  42. & The effect of chronic subarachnoid hemorrhage on basal endothelium-derived relaxing factor activity in intrathecal cerebral arteries. Journal of Neurosurgery 76
  43. & Impaired cyclic nucleotide-mediated vasorelaxation may contribute to closure of the human umbilical artery after birth. British Journal of Pharmacology 106(2), 348-353.
  44. & Blood flow and optimal vascular topography: role of the endothelium.. Basic Research in Cardiology 86 Suppl 2
  45. & Basal EDRF activity helps to keep the geometrical configuration of arterial bifurcations close to the murray optimum. Journal of Theoretical Biology 146(4), 545-573.
  46. & Myogenic autoregulation of flow may be inversely related to endothelium-derived relaxing factor activity. American Journal of Physiology - Heart and Circulatory Physiology 258
  47. & EDRF in intact vascular networks. Blood Vessels 27
  48. & Activities of endothelin-1 in the vascular network of the rabbit ear: A microangiographic study. British Journal of Pharmacology 101
  49. & Investigation of the vasoconstrictor action of subarachnoid haemoglobin in the pig cerebral circulation in vivo. British Journal of Pharmacology 97
  50. & EDRF-mediated dilatation in the rat isolated perfused kidney: A microangiographic study. British Journal of Pharmacology 98
  51. & The role of EDRF in flow distribution: A microangiographic study of the rabbit isolated ear. Microvascular Research 37(2), 162-177.
  52. & Endothelium-derived relaxing factor (EDRF) and resistance vessels in an intact vascular bed: a microangiographic study of the rabbit isolated ear. British Journal of Pharmacology 93(3), 654-662.
  53. & EDRF coordinates the behaviour of vascular resistance vessels. Nature 329
  54. & Unstimulated release of endothelium derived relaxing factor is independent of mitochondrial ATP generation. Cardiovascular Research 21
  55. & Haptoglobin-haemoglobin complex in human plasma inhibits endothelium dependent relaxation: Evidence that endothelium derived relaxing factor acts as a local autocoid. Cardiovascular Research 20
  56. & Influence of endothelium on drug-induced relaxation of the rabbit aorta. European Journal of Pharmacology 121(1), 19-23.
  57. & Production of endothelium derived relaxant factor is dependent on oxidative phosphorylation and extracellular calcium. Cardiovascular Research 20
  58. & Endothelium derived relaxant factor. Journal of the Royal College of Physicians of London 19
  59. & Endothelium influences coronary and aortic vasomotion by release of an unstable humoral factor.. Advances in myocardiology 6
  60. & Evidence that cyclic guanosine monophosphate (cGMP) mediates endothelium-dependent relaxation. European Journal of Pharmacology 112(2), 195-202.
  61. & Ergometrine-induced arterial dilatation: An endothelium-mediated effect. Journal of Molecular and Cellular Cardiology 16(5), 479-482.
  62. & Isolated perfused rabbit coronary artery and aortic strip preparations: the role of endothelium-derived relaxant factor.. The Journal of Physiology 351(1), 13-24.
  63. & The nature of endothelium-derived vascular relaxant factor. Nature 308(5960), 645-647.

Conference Contributions

  1. & (1990). Nitric oxide in arterial networks. Presented at Nitric oxide from L-arginine: a bioregulatory system: proceedings of a Symposium on Biological Importance of Nitric Oxide. ICS897,

Other Research Outputs

  1. Characterisation of cardiomyocytes derived from human induced pluripotent stem cells as monolayers. The Lancet
  2. A Systematized Approach to Investigate Ca2+ Synchronization in iPSC-Derived Cardiomyocyte Networks. Biophysical Journal